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However, glaucoma often develops with normal IOP and is regarded as normal in U2010tension glaucoma. The present research investigated the phosphorylation of collapsin response mediator protein (u20102), a protein that is abundantly present in neurons and plays a role in their development. We reported that the decrease in the number of RGCs and thickness of the inner retinal layer may have been minimized after NMDA treatment in CRMP2KI/KI mice with genetically impaired CRMP2 phosphorylation was genetically impaired CRMP2 phosphorylation. We investigated GLAST heterozygotes in CRMP2KI/KI and GLAST knockout mice with CRMP2KI/KI and GLAST knockout mice and compared them with GLAST+/u2212 mice and GLAST2112/u2212 mice.
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