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Normal-tension Glaucoma - Wiley Online Library

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Last Updated: 25 June 2022

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Genetic inhibition of collapsin response mediator protein‐2 phosphorylation ameliorates retinal ganglion cell death in normal‐tension glaucoma models

However, glaucoma often develops with normal IOP and is regarded as normal in U2010tension glaucoma. The present research investigated the phosphorylation of collapsin response mediator protein (u20102), a protein that is abundantly present in neurons and plays a role in their development. We reported that the decrease in the number of RGCs and thickness of the inner retinal layer may have been minimized after NMDA treatment in CRMP2KI/KI mice with genetically impaired CRMP2 phosphorylation was genetically impaired CRMP2 phosphorylation. We investigated GLAST heterozygotes in CRMP2KI/KI and GLAST knockout mice with CRMP2KI/KI and GLAST knockout mice and compared them with GLAST+/u2212 mice and GLAST2112/u2212 mice.

Source link: https://onlinelibrary.wiley.com/doi/10.1111/gtc.12971

* Please keep in mind that all text is summarized by machine, we do not bear any responsibility, and you should always check original source before taking any actions

* Please keep in mind that all text is summarized by machine, we do not bear any responsibility, and you should always check original source before taking any actions