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Methionine Sulfoxide - Springer Nature

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Last Updated: 13 May 2022

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Ectopic Expression of Maize Plastidic Methionine Sulfoxide Reductase ZmMSRB1 Enhances Salinity Stress Tolerance in Arabidopsis thaliana

Plant methionine sulfoxide reductases can restore oxidative damage done to intracellular proteins and, therefore, play a vital role in the response to abiotic stress. In a previous report, we found that ZmMSRB1 can be triggered by salinity stress. MSRB1's characterization, as well as lines with ectopic expression of MSRB1, suggested that MSRB1 contributes to salinity stress tolerance. Overexpression of ZmMSRB1 in Arabidopsis seedlings greatly reduced reactive oxygen species buildup, leading to the downregulation of ROS-producing genes and upregulation of ROS-scavenging genes, resulting in a significant rise in ROS-scavenging protein production and a significant rise in ROS-scavenging protein synthesis, resulting in a significant rise in ROS-scavenging protein synthesis and a significant rise in chemis seedling protein production and stadoxoodu s, which led to s, leading to the downregulation of ROSca coding and the downregulation of ROScavenging genes and s and s, a s and s and scavenging protein a When GSH synthesis was interrupted, the ZmMSRB1-induced response to salinity stress was partially impaired. Together, the results of the current study show that maize MSRB1 increases salinity resistance by regulating Na+/K+ transport, soluble sugar content, and ROS levels in A. thaliana.

Source link: https://doi.org/10.1007/s11105-021-01320-8


Metabolic benefits of methionine restriction in adult mice do not require functional methionine sulfoxide reductase A (MsrA)

Although there are growing signs that methionine redox regulation of certain aspects of cell function, cell function control is becoming more accessible, cellular interactions with MR remain largely unexplored. We investigated the effect of MR in mice on mice's metabolic benefits of the ubiquitously expressed methionine repair enzyme methionine reductase A. We investigated the extent to which MsrA is required for metabolic effects of MR in adult mice lacking MsrA. Overall, our results show that MsrA is not required for the metabolic benefits of MR in adult mice.

Source link: https://doi.org/10.1038/s41598-022-08978-4

* Please keep in mind that all text is summarized by machine, we do not bear any responsibility, and you should always check original source before taking any actions

* Please keep in mind that all text is summarized by machine, we do not bear any responsibility, and you should always check original source before taking any actions