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"However, a lack of research on the receptor-mediated EV-D68 entry into host cells has hindered our understanding of the tissue tropism and pathogenesis of EV-D68. " Cell surface sialic acid is required for EV-D68 to bind to and infect vulnerable cells, according to this research. Crystal structures of EV-D68 in a complex with sialylated glycan receptor analogues show that they bind to the virus surface in u2018canyon's u2019.
Source link: https://www.osti.gov/biblio/1239678
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