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Heterosis refers to the hybrid's outstanding results in comparison to its parental lines. Despite numerous genetic models and novel molecular pathways that have been developed to explain heterosis, it's unclear how cells can promote heterotic growth via complementary gene expression, metabolic accumulation, and/or hormone signaling. We discovered an increased TORC1 signaling of the elite hybrid rice Shanyou 63 relative to the parental lines, which was associated with increased growth-promoting and energy metabolic enzyme production, as well as improved panic cell growth. Metabolism of nuclear-cytosolic acetyl-Coenzyme A was also enhanced in the hybrid, which coincided with rises of histone H3 acetylation to selectively target growth-promoting and metabolic gene expressions. The results showed that an enhanced investment in hybrids would propel and maintain the TOR signaling feedback loop to encourage high growth, which may be a general mechanistic model for heterosis.
Source link: https://www.ncbi.nlm.nih.gov/bioproject/860563
Nitrate is a key molecular marker for plant adaptation to changing environmental conditions, but how nitrate regulates plant growth under carbon deficiency conditions is unclear.
Source link: https://www.ncbi.nlm.nih.gov/bioproject/852347
In an ideal and high-fat diet context, the aim of this research was to determine the effects of maternal vitamin D deficiency on adipose tissue metabolism and energy homeostasis in offspring, taking into account sex-specific responses. In juvenile male and female offspring of VDD mice, body weight, energy consumption, and spontaneous exercise were all different. In adulthood, a HF diet supplemented glucose homeostasis and adiposity in male offspring but not in females. Hence, maternal VDD sex-dependently modified the metabolic fate of the offspring, particularly when dealing with a HF diet in adulthood.
Source link: https://www.ncbi.nlm.nih.gov/bioproject/850402
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