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Cardiomyocytes Apoptosis - U.S. Department of Veterans Affairs

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Last Updated: 23 October 2021

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Nogo-A knockdown inhibits hypoxia/reoxygenation-induced activation of mitochondrial-dependent apoptosis in cardiomyocytes.

Configured cell death of cardiomyocytes adhering to myocardial ischemia boosts biomechanical anxiety on the remaining myocardium, bring about myocardial disorder that might lead to congestive heart failure or premature death. In this research, Nogo-A expression was shown to be significantly enhanced in heart cells from patients with dilated cardiomyopathy and from patients who have experienced an ischemic event. In arrangement with the findings from human tissue, Nogo-A expression was considerably increased in cultured neonatal rat cardiomyocytes based on hypoxia/reoxygenation.

Source link: https://doi.org/10.1016/j.yjmcc.2011.03.004

* Please keep in mind that all text is summarized by machine, we do not bear any responsibility, and you should always check original source before taking any actions

* Please keep in mind that all text is summarized by machine, we do not bear any responsibility, and you should always check original source before taking any actions