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Abstract Background: Patients with cardiac amyloidosis have an expanded and dysfunctional left atrium as a result of left ventricular diastolic and then systolic dysfunction, as well as amyloid infiltration into the LA wall. In CA, a single study revealed impaired LA strain, but the causes of reduced LA strain have not been established. The LA strain was much more impaired in patients with ATTR-CA than those without CA, and to a lesser degree than those with AL-CA. zo 0. 401, rho 0. 001, p=0. 042, t=0. 042, p=0. 001 ; 0. 001 vs. LV GLS In patients with AL-CA: 4C-PALS vs. RP 0. 041, rho 0. 241, rho 0. 401, t 0. 001, p=0. 042, p=0. 042 p=0. 042 p rho t rho 0. 141, 0. 11 rho 0. 051 0. 041, rho 0. 141 rho 0 0. 001 rho v vs. Patients with AL-CA appear to have greater relationships between LA strain, size, and haemodynamic load, perhaps representing the most acute disease course and a shorter time for amyloid deposition in the LA wall.
Source link: https://doi.org/10.1093/eurheartj/ehab724.1794
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