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D2CO3 crystallizes in the monoclinic space group P21/c, c = 5. 690, a= 5. 690, u00b0, Z = 4, with one symmetry-inequivalent anti-anti shaped D2CO3 molecule forming dimers, as previously predicted.
Source link: https://doi.org/10.3390/inorganics10090132
Carbon dioxide injection into a deep saline aquifer will lead to dissolving into formation brine and producing carbonic acid. The effect of these fluid-rock geochemical reactions on porosity, permeability, and multiphase flow responses of deep saline aquifers is important in determining the CO2 storage capacity of deep saline aquifers. In this study, carbonic acid flooding experiments were carried out on core samples consisting of poorly separated, quartz-rich sand with laminated bedding from a potential CO2 storage target in northwest Wyoming. To be more specific, within the more pore-permeable section of core samples with higher pore size, the permeability increase is apparent due to dolomite mineral grains and cements dissolution. Fluid-rock reactions' effects on changes in static and multiphase flow characteristics of eolian sandstones, which provide a comprehensive basis for more accurate prediction/simulation of CO2 injection into deep saline aquifers. This research provides a foundation for a more complete investigation/simulation of CO2 injection into deep saline aquifers.
Source link: https://doi.org/10.2118/212272-pa
Carbonic acid's apparent first dissociation constant has been established in various ways in the literature. HmHCO3/mCO2. In contrast, Hastings and Sendroy have established a clear constant in which acidity is expressed as H ion production: K'1 = aHmHCO3/mCO2. Specifically, for the derivation of bicarbonate concentration from PCO2 and pH, pK'1 is to be used, not uncorrected pKs.
Source link: https://doi.org/10.1152/ajpcell.1991.260.5.c1113
We analyzed arterial blood gases from CA II-deficient and normal control mice to determine the role of carbonic anhydrase II in the pulmonary CO 2 exchange. NaHCO 3 was intraperitoneally administered, and arterial blood gases were analyzed 4 hours later to determine the effect of metabolic acidosis on arterial blood gases. The metabolic acidosis in CA II-deficient mice was reduced, but respiratory acidosis became more severe. It is most likely that CO2 retention in these animals is due to CA II deficiency in both red blood cells and type II pneumocytes, according to the most.
Source link: https://doi.org/10.1152/ajplung.1998.274.2.l301
pH values for in situ pH remained significantly higher than in testicular artery or systemic arterial blood, according to the proximal tubules, proximal caput, middle corpus, and proximal cauda epididymidis, and did not change significantly after acetazolamide. In all buildings except the MCR, Acetazolamide's increased in situ PCO2 increased significantly. The total CO2 content in normal ST fluid was significantly higher than in a "primary" fluid, and both values were well below TA or SAB total CO2 levels.
Source link: https://doi.org/10.1152/ajpendo.1990.258.1.e143
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