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Carbon Monoxide Poisoning - Crossref

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Last Updated: 28 August 2022

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Severe carbon monoxide poisonings in scuba divers: Asia-Pacific cases and causation

There are reports of fatal incidents in recreational scuba divers as a result of carbon monoxide poisoning. Aim: There are reports of fatal events in recreational scuba divers. Methods: A hand search of Project Stickybeak reports, subsequent Australian fatality series studies, and of published New Zealand diving fatality reports and associated statistics were conducted, as well as key word searches of the National Coronial Information System for scuba fatalities in Australia and New Zealand. Cases associated with the Australasian Diving Safety Foundation's diving fatality database were found. One report from each of Australia, New Zealand, Singapore, and the Maldives found four scuba deaths as a result of CO poisoning. Scuba fatalities from CO poisoning are rare, but most likely under-reported. CO emissions entering the compressor and CO production by pyrolysis or gasification within the compressor or its filter system are among the CO sources.

Source link: https://doi.org/10.22462/05.06.2022.8


Evaluation of the Cardioprotective Effect of Granulocyte Colony Stimulating Factor in Patients with Carbon Monoxide Poisoning

In previous studies, the Granulocyte colony-stimulating factor on infarcted hearts was demonstrated. In this review, the effect of G-CSF on cardiac function of patients with moderate to severe acute CO poisoning was explored. G-CSF and normal saline were administered separately to treatment and placebo groups in each of the treatment and placebo groups. The findings revealed that myocardial injury is normal in moderate to severe CO poisoning. After G-CSF treatment, the frequency of ST depression, inversion, or flatt of T wave and QTC in ECG were significantly reduced. In addition, the incidence of cardiac arrhythmias due to CO poisoning was reduced after G-CSF therapy. In CO-poisoned patients, however, G-CSF did not have any protective effects on the TPI level and function of the left ventricular. Conclusion: GCSF may possibly reduce CO-induced cardiac ischemia in patients with acute CO poisoning.

Source link: https://doi.org/10.2174/0929866527666201022112810


Carbon Monoxide Poisoning

Abstract — a family of traveling carnival workers bought a new trailer about a family's holiday to a new cabin They awakened one morning sick and vomiting and discovered their young son was unconscious. The linguistic analysis reviewed the risk arguments put forth by the American National Institute with the owner's manual that came with their generator, as well as a similar owner's manual of a generator that they did not buy compared to others.

Source link: https://doi.org/10.1093/acprof:oso/9780195328837.003.0010


Release of glutathione from erythrocytes and other markers of oxidative stress in carbon monoxide poisoning

Gluathione is released from erythrocytes and other signs of oxidative stress in carbon monoxide poisoning by oxidative stress. 82: 1424u20131432, 1997. u2014Rats exposed to CO in a way that can cause oxidative stress in the brain resulted in a twofold rise in plasma levels of oxidized proteins, thiobarbituric acid-reactive substances, oxidized glutathione, and reduced glutathione, despite a twofold increase in oxidized proteins, thiobarbituric acid-reactive substances, 82 GSH and GSSG did not rise in GSH and GSSG after exchange transfusion with a hemoglobin-containing saline solution, suggesting that blood-borne cells secretly released these peptides in response to oxidative stress. The results show that nitric oxide-derived oxidants are involved in CO-mediated oxidative stress within the vascular compartment, and that elevations of several compounds may be useful in determining CO-mediated respiratory stress within the vascular compartment, as well as brain injury risks.

Source link: https://doi.org/10.1152/jappl.1997.82.5.1424


Voxel-Based and Surface-Based Morphometry Study of the Early Stage of Carbon Monoxide Poisoning-Induced Parkinsonism

In previous studies, grey matter volume changes in carbon monoxide poisoning-induced parkinsonism have been investigated. VBM's report showed decreased GM volumes in the bilateral frontal and temporal lobes, anterior cingulate cortex, amygdala, thalamus, and cerebellum, as well as a trend towards WM atrophy in the anterior cerebral and medial mesencephalon, although in the posterior cerebral and motor cortical regions. The patients' left supramarginal gyrus showed widespread cortical thinning that extended beyond the frontotemporal areas and involved the occipitoparietal areas and regional shallow sulcal depth in the left supramarginal gyrus. Nonmotor disorders were correlated with atrophy in the medial orbital superior frontal gyrus, ACC, caudate, thalamus, and caudal middle frontal gyrus, and caudal middle frontal gyrus, according to GM's atrophy in the medial orbital superior frontal gyrus, ACC, thalamus, amygdala, and cerebellum crus I.

Source link: https://doi.org/10.21203/rs.3.rs-1777825/v1


Carbon Monoxide Poisoning in the Veterans Health Administration, 2010 - 2016

This study was the first to focus on CO poisoning trends in the VHA population. MethodsQueries were conducted in the VA PraedicoTMPublic HealthSurveillance System for inpatient discharges and emergency roomoutpatient visits with ICD 9/10 CM codes for CO poisoning from 1/1/2010 to June 30, 2016. Results: For the same timeframe, patients with carboxyhemoglobin blood level measurements (u226510%2 were extracted and merged with the CO poisoning registry, we calculated estimates using total unique users of VHA care formatching time frame and geographic location as denominators. The overall incidence of CO poisoning in the study period was 18 per million unique users of VHA care. Patients with the diagnosis of accidental poisoning had the largest percentage of patients seen in the ED and later admitted, with 36 unique patients. In 111 of patients with COHb bloodlevels>u226510% were present in 111 of patients with CO poisoningcodes, the COHb blood levels reached u226510%. Both patients with COHb measures were found to be less than half, those with self-harm classification were least well represented with only 7 unique patients. According to results, CO poisoning in the VHA patient population has not been well studied. Given the low percentage of cases with COHb blood test results, further study into investment is required in VHA.

Source link: https://doi.org/10.5210/ojphi.v9i1.7710


"Xingnao Kaiqiao" Acupuncture Combined with Traditional Chinese Medicine for Delayed Encephalopathy after Carbon Monoxide Poisoning: A Case Report

Purpose: To investigate the clinical effects of acupuncture combined with traditional Chinese medicine in the treatment of delayed encephalopathy of carbon monoxide poisoning, we'll look at the treatment of delayed encephalopathy of carbon monoxide poisoning by traditional Chinese medicine. Conclusion: The findings of this study reveal that acupuncture combined with traditional Chinese medicine is safe in the prevention of carbon monoxide poisoning delayed encephalopathy, with the benefits of a short course of treatment and integration of diagnosis and prevention.

Source link: https://doi.org/10.46633/gjn.030103


Rare Causes of Acute Coronary Syndrome: Carbon Monoxide Poisoning

CO poisoning can cause a variety of cardiovascular disorders, including ACS. We've done a retrospective review in the Toxicology Department of Saint Spiridon Emergency University Hospital, including all patients admitted via CO poisoning in the emergency department. Compared to the non-event group, Cardiac enzyme markers showed a statistically significant rise in the event group. Intoxication in the STEMI and NSTEMI groups was evident. Any type of poisoning, myocardial injury can result in CO poisoning, and it can be temporary, reversible, or permanent. Our investigation, which is focusing on the ACS, uncovers new data on adverse cardiac events in patients with CO poisoning. Because 50% of patients in the event group were heavily intoxicated, we found that the severity of CO poisoning plays a vital role in myocardial disease.

Source link: https://doi.org/10.3390/life12081158


Pediatric Carbon Monoxide Poisoning

Since humans invented carbon monoxide-based combustion products, human history has been documented. The Romans understood that CO poisoning leads to death. Carbon monoxide is a colorless, tasteless, odorless gas by-product of the combustion of carbon-containing compounds such as natural gas, oil, kerosene, propane, and charcoal. Carbon monoxide poisoning can also occur from space heaters, methylene chloride in paint removers, and fire. In the United States, carbon monoxide is the most common cause of poisoning injury and death worldwide, as well as accidental and intentional poisoning. About 40,000 emergency department visits and 800 deaths per year in the United States due to carbon monoxide poisoning results. Children under the age of 4 years have the highest incidence of accidental CO poisoning among intentional CO poisoning, but the lowest death rates have been found.

Source link: https://doi.org/10.1093/oso/9780195342680.003.0073


Management of carbon monoxide poisoning

CO poisoning is the most common cause of toxic death. When people inhale CO, the gas diffuses quickly to the body and replaces oxygen at the level of haemoglobin, myoglobin, and other oxygen carriers. Following CO poisoning, a number of signs may appear. Survivors of CO poisoning often have recurrent neurological sequelaes or delayed neurological signs. There is no correlation between carboxyhemoglobin levels and clinical signs.

Source link: https://doi.org/10.1093/med/9780199600830.003.0328

* Please keep in mind that all text is summarized by machine, we do not bear any responsibility, and you should always check original source before taking any actions

* Please keep in mind that all text is summarized by machine, we do not bear any responsibility, and you should always check original source before taking any actions