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Air Pollution - Europe PMC

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Last Updated: 08 May 2022

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What Is "Socioeconomic Position (SEP)," and How Might It Modify Air Pollution-Health Associations? Cohering Findings, Identifying Challenges, and Disentangling Effects of SEP and Race in US City Settings.

Purpose of study Environmental epidemiology has long considered socioeconomic status to be a key predictor of pollution risks in the United States, given that lower-income and minority groups are often exposed to pollution in the United States. A growing body of literature has shown that lower-SEP groups may also be more vulnerable to pollution in recent decades. In the urban U. S. context, it is now extremely difficult to distinguish SEP from race. Here, we cohere data from our latest and on-going studies aimed at identifying synergistic health effects among SEP-related stressors and pollutants. We provide a glance at the complex interplay among SEP, community stressors, race, and pollution in U. S. cities, combining quantitative findings with qualitative data on neighborhood perceptions and stress.

Source link: https://europepmc.org/article/MED/35511352


Effect of traffic-related air pollution on cough in adults with polymorphisms in several cough-related genes.

People with specific genetic predispositions and sensitivities are at a higher risk of experiencing respiratory problems, particularly chronic cough, as a result of widespread global air pollution. We investigated whether a genetic risk score based on cough-related single-nucleotide polymorphisms is associated with a cough count greater than 24 hours post-exposure to diesel exhaust, a measure of traffic-related air pollution. We discovered that GRS produced from 7 SNPs related to TRPA1, TRPV1, and NK-2R were correlated with cough count under closely monitored conditions. Following DE exposure and filtered air exposure, we found a significant, positive correlation between GRS and cough count, which was a positive result. In summary, cough-relevant GRS was correlated with a higher 24 h cough rate in a controlled environment, meaning that individuals with a high GRS may be more susceptible to cough regardless of their exposure.

Source link: https://europepmc.org/article/MED/35509099


Air pollution, residential greenness, and metabolic dysfunction biomarkers: analyses in the Chinese Longitudinal Healthy Longevity Survey.

We investigated the impact of fine particulate matter and the Normalized Difference Vegetation Index on MetS, both locally and online. Ambient air pollution levels were similar between rural and urban areas. Both the cross-sectional and longitudinal study showed promising links between PM 2. 5 and common abdominal obesity and MetS, as well as a negative relationship of NDVI with persistent AO. PM 2. 5 and NDVI had a statistically significant relationship on AO prevalence, with PM 2. 5 and NDVI having a statistically significant correlation on AO prevalence. The link between PM 2. 5 and MetS, AO, elevated fasting glucose, and reduced HDL-C was only relevant in rural areas, not in urban areas. NDVI and AO was only relevant in areas with low PM 2. 5 or worse than high PM 2. 5. These study findings recommend for green space planning in urban areas and air pollution reduction in rural areas.

Source link: https://europepmc.org/article/MED/35509051


Air Purification Process from CO 2 Pollution by Considering Porous Iron Membrane Performance: The Molecular Dynamics Approach

We explore the porous iron matrix as a metallic membrane for air purification from Carbon dioxide pollution using a molecular dynamics simulation approach in recent computational studies. Temperature/pressure changes from 300 K/1 bar to 350 K/5 bar in MD simulations are affected by this process, and the number of absorbed CO 2 molecules in MD simulations varies from 121 to 93/145 molecules. After CO 2 molecules separation from O2 molecules with porous iron matrix, Young's porous iron membrane module drops from 5. 83 GPa to 5. 30 GPa.

Source link: https://europepmc.org/article/PPR/PPR488198


Increased allostatic load associated with ambient air pollution acting as a stressor: Cross-sectional evidence from the China multi-ethnic cohort study.

Allostatic load refers to the cumulative biological burden caused by chronic stressors. Air pollution acting as a stressor initiates the neuroendocrine system and then leads to multi-organ dysfunction, which ultimately leads to allostatic load. Emerging experimental evidence shows that air pollution acting as a stressor activates the neuroendocrine system and then results in multi-organ dysfunction, leading to allostatic load. We want to investigate the correlations between chronic exposure to air pollution and allostatic load in Chinese adults. A satellite-based random forest strategy determined ambient air pollution levels. According to an elevated risk of allostatic load, long-term exposure to ambient air pollution was strongly linked to an elevated risk of allostatic load, with relative risk of 1. 040, 1. 029, and 1. 087 for each 10 g/m3 rise in ambient PM2. 5, PM10, and O3, respectively. Some intrinsic causes and non-chemical stressors can influence the correlations between air pollution and allostatic load. People with older, minority, lower education, and lower income were allostatic loads, owing to a significant rise in air pollution.

Source link: https://europepmc.org/article/MED/35523330


Associations of long-term exposure to ambient air pollution and road traffic noise with sleep health in UK Biobank.

Background Information The connection between pollutants and sleep is limited, and the majority of studies concentrated on individual sleep habits, not interrelation. We wanted to investigate the connection between ambient air pollution and road traffic noise, which was linked to overall sleep quality. To create a general sleep pattern, you'll need five sleep habits in addition to five sleep habits. Using the Land Use Regression model, the estimated risk of ambient air pollution was estimated. Using a simplified version of the Common Noise Assessment Methods model, road traffic noise exposure was estimated. Poor sleep pattern: 1. 13 % for intermediate: 1. 03 ; for intermediate: 1. 03 ]. Long-term ambient air pollution is attributed to general sleep quality, according to studies. Road traffic noise itself is less correlated with overall sleep quality.

Source link: https://europepmc.org/article/MED/35513113


Associations between outdoor air pollution, ambient temperature and fraction of exhaled nitric oxide (FeNO) in university students in northern China - A panel study.

In university students in northern China, Aim To determine the relationships between ambient temperature, air pollution, and FeNO. Methods We conducted a panel study in 67 university students without asthma treatment in Taiyuan, Taiwan's capital. NO 2, NO 2, and O 3 were measured 7 days before the FeNO experiment was conducted. For O 3 and 0. 93-3. 14 mg/m 3 for CO, the range of lag 7 pollution was 105. 0-339. 0 g/m 3 for PM 10, 36. 0-187. 0 g/m 3 for PM 2. 5, 36. 0-187. 0 m 3 for PM 2. 5, 36. 0-3,0 g/m 3 was 105. 0-339. 0 g/m 3 for SO 2 and 32. 0-367. 0 g/m 3 was 105. 0-339. Both men and women had similar experiences with FeNO due to exposure and pollution. Conclusion PM 10, PM 2. 5, and outdoor temperature can be associated with airway inflammation in young adults in northern China, measured as FeNO, while CO can be negatively associated with FeNO.

Source link: https://europepmc.org/article/MED/35513063


Toxicological effects of traffic-related air pollution on the lungs: Evidence, biomarkers and intervention.

Background Numerous epidemiological studies have reported that exposure to traffic-related air pollution elevates the risk of various respiratory diseases. Output: The airway hyperresponsiveness indicated the first evidence of a lung injury caused by GEP exposure. GEP exposure significantly enhanced the airway resistances and reduced lung compliance in comparison to the control group, as the greater the dose of GEP was, the more serious the lung injury is. The increase of inflammatory cells, including the lymphocytes and neutrophils, in the BAL fluid, revealed lung injury in the Lung tissue. Histological changes in the lung tissues were even more apparent as a result of increased GEP dose: inflammatory cell infiltration increased and alveolar wall thickened. Conclusion The toxicological effects of traffic-related air pollution on rat lungs are demonstrated by evidence and biomarkers, and vitamin E therapy is also feasible.

Source link: https://europepmc.org/article/MED/35512471


Impact of air pollution on outdoor cultural heritage objects and decoding the role of particulate matter: a critical review.

Both Atmospheric damage and soiling are recognized as two key aspects of cultural heritage material degradation resulting from air pollution. However, the PM's atmospheric damage effect is more complex; its variable composition accelerates the degradation process. One of the main areas of current research is to review the existing evidence on PM influence on atmospheric damage, in order to better understand and critically assess the key areas in current knowledge. Soiling is the second phenomenon that is attributed to cultural heritage information and PM pollution. In the past, black carbon was believed to be the key soiling agent, and that the change of the lightness could accurately measure soiling. Despite numerous studies addressing soiling and atmospheric damage separately, there is an overlap between these two processes. No systematic studies have been published on the relationship between soiling and atmospheric damage caused by atmospheric PM.

Source link: https://europepmc.org/article/MED/35501442


Identification of Health Effects of Complex Air Pollution in China

As the development of air pollution control in China has lowered, the amount of particulate matter has decreased, but the level of ozone has grown, posing a significant danger to public health. The air pollutant constituent condition is broken into eight categories based on different combinations of PM 2. 5, NO 2, and O 3 concentrations. PM 2. 5, O 3, and NO 2 and type 4 with higher PM 2. 5 and NO 2 have a higher risk of relative harm among them. The effect of air pollution interactions in the test indicated that the excess risk of multi-pollutants was less than the straightforward sum of individual air pollution causes, although partially incorrect conclusions could have been drawn ignoring the presence of interactions between air pollutants.

Source link: https://europepmc.org/article/PPR/PPR487469

* Please keep in mind that all text is summarized by machine, we do not bear any responsibility, and you should always check original source before taking any actions

* Please keep in mind that all text is summarized by machine, we do not bear any responsibility, and you should always check original source before taking any actions