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It has long been known that aerobic exercise aids in preventing and improving non-alcoholic fatty liver. However, it is also unknown if aerobic exercise can prevent and treat hepatic lipid accumulation via SRA. The mice were randomly divided into four groups as follows: normal control group, ordinary aerobic exercise group, high-fat diet group, and high-fat diet plus aerobic exercise group were randomly divided into four groups, as follows: normal control group, normal aerobic exercise group, high-fat diet group, and high-fat diet plus aerobic exercise group. In the HAE group, body weight, as well as blood TC, LDL-C, and liver TG were significantly lower than in the HFD group than in the HFD group.
Induction of sepsis, mice receiving 5 mg/kg LPS and Ex + LPS were intraperitoneally administered sepsis. This review investigated the effects of AE on systemic inflammation, lung permeability, and bronchoalveolar fibrous fluid cell count, oxidative stress-related variables, plasma glucose levels, serum insulin levels, plasma glucose level, and plasma high-mobility group box 1 results, as well as nuclear factor erythroid 2-related factor 2 mRNA expression levels in lung tissue. Neuophil content in organs has been reduced by AE by a large amount. During sepsis, AE reduced the serum levels of lactate and HMGB1, but serum glucose levels and serum insulin levels increased. AE impairs LPS-induced lactate and HMGB1 for a portion because AE elevates serum insulin levels and reduces the amounts of Glut1. AE is a novel therapeutic approach for sepsis affecting aerobic glycolysis.
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