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The absence of single-cell examinations into the broader population of patients with community-acquired pneumonia renders it tough to identify immune features special to COVID-19 from the common features of a dysregulated host response to pneumonia. With this well balanced, multi-omic technique we define splitting transcriptional and phenotypic patterns in T cell, NK cell and monocyte populations between patients with CAP brought on by either SARS-CoV-2, Influenza A or various other pathogens, and thus expand our understanding of the outer immune response in various forms of CAP.
Source link: https://www.ncbi.nlm.nih.gov/bioproject/692496
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